By Moore R. L.
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Additional info for An Extension of the Theorem That No Countable Point Set Is Perfect
These problems reflect the assumption that selection alone explains the feature of interest (Gould and Lewontin 1979). But even if a general skep ticism about optimization is not justified, these models contributed little to the debate about the etiology of NoRs that emerged in the 1980s. Plasticity as a By-product of Selection As Via (1994) noted, when Bradshaw published his review in 1965, “there were no population genetic models for the evolution of quantitative traits under natural selec tion, even in single environments” (p.
Each organism is plastic in at least some traits—typically in many traits—and almost all traits are plastic in at least some organ isms. Plasticity is thus pervasive and exists in many different forms. The only thing that all plasticities have in common is that the phenotype is induced by the environment; in other words, the environment acts as a cue to form the phenotype. Two different processes, described by Smith-Gill (1983), may be involved in plasticity. Phenotypic modulation is the simple case.
The use of rG values is known as the character state approach. An alternative, function approach is based on the idea that a plastic phenotype can be seen as a function of the environment. In this approach, one typically estimates genetic parameters for a function describing continuous reaction norms. One can, for example, estimate the heritability for the slope in case of linear reaction norms, as an estimate of the heritability of plasticity (Scheiner and Lyman 1989). 2 for a simu lated example).
An Extension of the Theorem That No Countable Point Set Is Perfect by Moore R. L.